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Researchers in China identify new tick-borne virus linked to flu-like symptoms and organ inflammation

A study published in the New England Journal of Medicine found the newly discovered virus in over 10% of tested patients bitten by ticks.

The short version

  • Scientists identified the Asian longhorned tick nairovirus (ALTNV) after screening blood samples from thousands of patients with unexplained illnesses following tick bites.
  • The virus causes flu-like symptoms, low blood platelet counts, and liver-cell inflammation, but was not linked to fatalities on its own.
  • Coinfection with both ALTNV and Dabie bandavirus resulted in severe complications and seven deaths during the study.
  • No specific antiviral treatments or vaccines exist for ALTNV, leaving clinicians reliant on supportive care and accurate differential diagnosis.

Key facts

  • Researchers from the State Key Laboratory of Pathogen and Biosecurity in Beijing identified ALTNV in 10.4% of 3,163 patients who had sustained tick bites.[Fox News]
  • ALTNV infection causes fatigue, gastrointestinal issues, flu-like symptoms, low platelet counts (thrombocytopenia), and liver-cell inflammation.[Fox News]
  • Among patients coinfected with both ALTNV and Dabie bandavirus (DBV), respiratory and kidney complications were more prevalent, and seven individuals (18.4%) died.[Fox News]
  • The research findings on ALTNV were published in the New England Journal of Medicine.[Fox News]
  • There are currently no vaccines or targeted antiviral drugs available for ALTNV, making supportive medical care the standard course of treatment.[Fox News]

What remains uncertain

  • The broader geographic spread of ALTNV outside the tested patient cohort in China remains unmeasured.[Fox News]
  • The exact biological mechanisms causing heightened severity and mortality in patients coinfected with both ALTNV and DBV have not been fully determined.[Fox News]

Sources

Outlet counts describe coverage, not independent confirmation. Reports may share a wire service or original source.